| protein
| interacting with THAP1, THAP3 and thus involved in cell proliferation  |
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HCFC1 is required for stabilizing OGT in the nucleus (interactions of OGT with multiple HCFC1 domains may indicate that OGT has several functions in association with HCFC1) |
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TAB1 is modified through OGT on a single site, Ser395 (this modification is induced by IL1 and osmotic stress, known inducers of the MAP3K7 signalling cascade) |
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associates with ligand-bound glucocorticoid receptor in a multi-protein repression complex |
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interacts with the tetratricopeptide repeat binding site of HSP90AA1 |
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TET2 and TET3 associate with OGT, an enzyme that by itself catalyses the addition of O-GlcNAc onto serine and threonine residues (O-GlcNAcylation) |
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OGT associates with TET2 at transcription start sites |
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TET1 and TET2 as stable partners of OGT in the nucleus of ESCs |
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link between TET1 and OGT activities in regulating CpG island methylation |
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POU2F1 integrates metabolic and stress signals via OGT modification to regulate target gene activity |
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OGT-modification of CAMK2A is a novel signalling event in pathways that may contribute critically to cardiac and neuronal pathophysiology in diabetes and other diseases |
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OGT is not only a major TET3-interacting protein but also regulates TET3 subcellular localization and enzymatic activity |
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mediates O-GlcNAcylation of the SNARE protein SNAP29 and regulates autophagy in a nutrient-dependent manner |
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OGT modifies and regulates an essential epigenetic tool, RNF2, which may contribute to embryonic stem cells (hESC) pluripotency maintenance and differentiation |
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distinct OGT-binding sites in HCFC1 promote proteolysis |
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KMT2E protein stability is cooperatively regulated by O-GlcNAc transferase (OGT) and ubiquitin-specific protease 7 (USP7) |
