| protein
| associating with IGBP1 |
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with GRM3(by Cterminal cytoplasmic tail) |
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PPP2CA involvement is a molecular mechanism for FCAR ligand binding regulation, a key step in initiating an immune response  |
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IGBP1 plays a required role in regulating the assembly and maintenance of adaptive PPP2CA phosphatase complexes |
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PPP2CA inhibiting IL23A production by suppressing the expression of the IL23A gene |
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in addition to PPP2CA, IGBP1 interacts with UBR5 and PABPC1, suggesting its involvement in multiple steps in the MTOR pathway that leads to translation initiation and cell-cycle progression |
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reduction of PPP2CA activity due to an enhanced IGBP1-PPP2CA interaction contributes directly to chemical carcinogen-induced tumorigenesis |
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STRN, STRN32, STRN4 binds directly to PPP2CA and PDCD10 |
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IGBP1 interacts with MID1, a microtubule-associated ubiquitin E3 ligase that appears to regulate the function of PPP2CA |
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SEMA3A elevates endothelial cell permeability through PPP2CA inactivation |
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GSK3B can inhibit PPP2CA by increasing the inhibitory L309-demethylation involving upregulation of PPME1 and inhibition of LCMT1 |
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CABP4 is dephosphorylated by PPP2CA in the retina |
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NMNAT2 affects MAPT phosphorylation by regulating PPP2CA activity |
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role for MID1 in the regulation of IGBP1 that is likely to impact the stability and activity level of PPP2CA |
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IGBP1 phosphoprotein binds to the protein phosphatase 2A catalytic subunit (PPP2CA) to regulate PP2A activity, and to poly(A)-binding protein (PABP) and progestin-inducible UBR5 |
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SIK2 attenuates the association of the PPP2CA repressor, the protein phosphatase methylesterase-1 (PPME1), thus preserving the methylation status of the PPP2CA catalytic subunit |
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in addition to recruiting PPP2CA to dephosphorylate cohesin and CDCA5, SGO1 physically shields cohesin from WAPL |
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MID1 catalyze the polyubiquitination of IGBP1, a protein regulator of protein phosphatase 2A (PPP2CA) |
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STK36 ubiquitination and cleavage is one of the key elements connecting the MID1-PPP2CA protein complex with GLI3 activity control |
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CIP2A interact with Protein phosphatase 2 (PPP2CA) to stabilize MYC and prevent its degradation |
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PPFIA1 functioned with PPP2CA to promote the dephosphorylation of KIF7, triggering KIF7 localization to the tips of primary cilia and promoting GLI transcriptional activity |
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PPME1 affects the activity of PPP2CA by demethylating PP2Ac, but also by directly binding to the phosphatase active site, suggesting loss of PPME1 in cells would enhance PPP2CA activity |
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a significant fraction of total PPP6C is associated with IGBP1, whereas a minimal fraction of total PPP2CA is associated with IGBP1 |
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RAB9A, competes with the catalytic subunit PPP2CA in binding to PPP2R1A, which has an important role in controlling the PPP2CA catalytic activity, compromised in several solid tumors and leukemias |
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regulation of protein phosphatase 2A (PPP2CA) tumor suppressor function by PPME1 |
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oncoprotein CIP2A is stabilized via interaction with tumor suppressor PPP2CA |
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PPP2CA regulated the stem cell factor (SCF)-induced activation of MAPK14/cofilin signaling pathway and subsequent migration of cardiac stem cells (CSCs) by interaction with MAPK14 |
