| Other morbid association(s)
|
| Type | Gene Modification | Chromosome rearrangement | Protein expression | Protein Function
|
|---|
| constitutional
|
|
| --over
|
| |
dramatically in cardiovascular accidents, cerebral trauma, brain tumors and creutzfeldt-jacob disease | | constitutional
|
|
| --over
|
|
early elevations (up to 3 days) of S100B and ENO2 secondary to severe traumatic brain injury, predict deterioration to brain death, but more prominently associated with ENO2 than S100B  | | constitutional
|
|
| --over
|
| |
in patients with migraine-without aura, increased ictal serum S100B and ENO2 levels together with increased interictal levels of S100B suggested that migraine might be associated with glial and/or neuronal damage in the brain and a prolonged disruption of blood-brain barrier | | constitutional
|
|
| --low
|
| |
associated with better neurologic outcomes of head trauma in children ( | | constitutional
|
|
| --over
|
| |
in tic disorders (TD) | | constitutional
|
|
| --over
|
| |
in diabetic ketoacidosis and correlated with the severity of hyperglycemia, ketosis, and acidosis | | constitutional
|
|
| --over
|
| |
in asymptomatic carriers of Leber hereditary optic neuropathy ( | |
