| 1 | BAZ1A, BAZ1B, SMARCA5
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| Non-canonical reader modules of BAZ1A promote recovery from DNA damage.
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| Oppikofer M, Sagolla M, Haley B, Zhang HM, Kummerfeld SK, Sudhamsu J, Flynn EM, Bai T, Zhang J, Ciferri C, Cochran AG.
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| Nat Commun 8(1):862. doi: 10.1038/s41467-017-00866-0.
2017
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| 2 | BAZ1B, MYO1C, SMARCA5
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| The chromatin remodelling complex B-WICH changes the chromatin structure and recruits histone acetyl-transferases to active rRNA genes.
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| Vintermist A, Böhm S, Sadeghifar F, Louvet E, Mansén A, Percipalle P, Ostlund Farrants AK.
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| PLoS One 6(4):e19184. doi: 10.1371/journal.pone.0019184.
2011
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| 3 | BAZ1B, H2AFX
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| WSTF regulates the H2A.X DNA damage response via a novel tyrosine kinase activity.
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| Xiao A, Li H, Shechter D, Ahn SH, Fabrizio LA, Erdjument-Bromage H, Ishibe-Murakami S, Wang B, Tempst P, Hofmann K, Patel DJ, Elledge SJ, Allis CD.
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| Nature 457(7225):57-62. Epub 2008 Dec 17.
2009
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| 4 | BAZ1B, MYO1C, SMARCA5
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| Distinct function of 2 chromatin remodeling complexes that share a common subunit, Williams syndrome transcription factor (WSTF).
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| Yoshimura K, Kitagawa H, Fujiki R, Tanabe M, Takezawa S, Takada I, Yamaoka I, Yonezawa M, Kondo T, Furutani Y, Yagi H, Yoshinaga S, Masuda T, Fukuda T, Yamamoto Y, Ebihara K, Li DY, Matsuoka R, Takeuchi JK, Matsumoto T, Kato S.
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| Proc Natl Acad Sci U S A 106(23):9280-5. Epub 2009 May 26.
2009
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| 5 | BAZ1B
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| Phosphorylation of Williams syndrome transcription factor by MAPK induces a switching between two distinct chromatin remodeling complexes.
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| Oya H, Yokoyama A, Yamaoka I, Fujiki R, Yonezawa M, Youn MY, Takada I, Kato S, Kitagawa H.
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| J Biol Chem 284(47):32472-82. Epub 2009 Sep 23.PMID: 19776015 2009
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| 6 | BAZ1B, SMARCA5
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| The WSTF-SNF2h chromatin remodeling complex interacts with several nuclear proteins in transcription.
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| Cavellán E, Asp P, Percipalle P, Farrants AK.
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| J Biol Chem 281(24):16264-71. Epub 2006 Apr 9. 2006
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| 7 | BAZ1B, MYO1C, SMARCA5
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| The chromatin remodelling complex WSTF-SNF2h interacts with nuclear myosin 1 and has a role in RNA polymerase I transcription.
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| Percipalle P, Fomproix N, Cavellán E, Voit R, Reimer G, Krüger T, Thyberg J, Scheer U, Grummt I, Farrants AK.
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| EMBO Rep 7(5):525-30. Epub 2006 Mar 3.
2006
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| 8 | BAZ1B, PCNA, SMARCA5
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| The Williams syndrome transcription factor interacts with PCNA to target chromatin remodelling by ISWI to replication foci.
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| Poot RA, Bozhenok L, van den Berg DL, Steffensen S, Ferreira F, Grimaldi M, Gilbert N, Ferreira J, Varga-Weisz PD.
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| Nat Cell Biol 6(12):1236-44. Epub 2004 Nov 14. 2004
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| 9 | BAZ1A, BAZ1B, BAZ2A, SMARCA5, TTF1, UBTF
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| NoRC--a novel member of mammalian ISWI-containing chromatin remodeling machines.
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| Strohner R, Nemeth A, Jansa P, Hofmann-Rohrer U, Santoro R, Langst G, Grummt I.
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| EMBO J 20(17):4892-900. 2001
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| 10 | BAZ1A, BAZ1B, BAZ2A, BAZ2B, BRD1, BRDT
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| A Novel Family of Bromodomain Genes.
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| Jones MH, Hamana N, Nezu Ji, Shimane M.
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| Genomics 63(1):40-45. 2000
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| 11 | BAZ1B, CPT1A, CPT1B, CPT1C, CPT2, CROT
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| Genomics of the human carnitine acyltransferase genes.
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| van der Leij FR, Huijkman NC, Boomsma C, Kuipers JR, Bartelds B.
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| Mol Genet Metab 71(1-2):139-53. 2000
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| 12 | BAZ1B
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| Structure of the PHD zinc finger from human Williams-Beuren syndrome transcription factor.
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| Pascual J, Martinez-Yamout M, Dyson HJ, Wright PE.
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| J Mol Biol 304(5):723-9. 2000
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| 13 | BAZ1B
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| ACF consists of two subunits, Acf1 and ISWI, that function cooperatively in the ATP-dependent catalysis of chromatin assembly.
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| Ito T, Levenstein ME, Fyodorov DV, Kutach AK, Kobayashi R, Kadonaga JT.
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| Genes Dev 13(12):1529-39. 1999
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| 14 | BAZ1B, D7S2476
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| Identification of the WBSCR9 gene, encoding a novel transcriptional regulator, in the Williams-Beuren syndrome deletion at 7q11.23.
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| Peoples RJ, et al.
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| Cytogenet Cell Genet 82 : 238-246. 1998
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| 15 | BAZ1B, WBS
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| A novel human gene, WSTF, is deleted in Williams syndrome.
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| Lu X, et al.
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| Genomics 54 : 241-249. 1998
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| 16 | BAZ1B
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| Chromatin-remodelling factor CHRAC contains the ATPases ISWI and topoisomerase II.
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| Varga-Weisz PD, Wilm M, Bonte E, Dumas K, Mann M, Becker PB.
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| Nature 388(6642):598-602. 1997
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