protein
| regulate TEAD1 and CEBPB transcriptional activity in the absence of environmental stress and suggests a role for MAPK14 in the expression of extracellular matrix components that maintain organ architecture |
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SP7 (Osterix) is a novel substrate for MAPK14, and Ser-73 and Ser-77 are the regulatory sites phosphorylated by MAPK14  |
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MAPKAPK2 and MAPK14 function in concert to phosphorylate Keratin 8, Keratin 18, and Keratin 20 in intestinal epithelia  |
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interacting with MYCBP2 (modulates TRPV1 desensitization through MAPK14 signaling which presents not only a novel model for the modulation of TRPV1-mediated signals but also expands the role of MAPK14 in the regulation of activity-induced receptor internalization)  |
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interaction between phosphorylated MAPK14 and cleaved N-terminal ATF6 could results in a protective effect against the vicious cycle of oxidative stress-ER stress by induction of ER chaperones and ERAD components  |
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CDK5RAP3 binds MAPK14, alters its cellular localization, and inhibits basal and cytokine-stimulated MAPK14 activity  |
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interplay between MAPK14 phosphorylation and DNAJC3 upregulation has key roles in modulating ERP29-induced cell-growth arrest and survival  |
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SMAD7 and MAPK14 mitogen-activated protein kinase together regulate the expression of APC and cell migration in prostate cancer cells in response to TGFB1 stimulation  |
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possible involvement of S100A12 in the development of osteoarthritis (OA) by up-regulating MMP13 and VEGFA via MAPK14 and NFKB1 pathways  |
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PDGFB-induced activation of MAPK14 was significantly suppressed by SERPINA12  |
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dual phosphorylation of PXN by JNK and MAPK14 is essential for T-cell activation, suggesting that NFATC1 is a functional target of the JNK/MAPK14 phosphorylated paxillin  |
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spatial regulation of MAPK14 by GADD45B/MAP3K4 negatively regulates the autophagic process  |
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OPRK1 activates MAPK14 through a phosphorylation and arrestin-dependent mechanism |
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cascade of posttranslational modifications involving MAPK14, KAT5, and MAPKAPK5, three proteins that are essential for RAS-induced senescence  |
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role for MAPK14 in mediating cross-talk between apical Na(+) entry via SCNN1G and its basolateral exit via ATP1A1, which may allow principal cells to maintain intracellular Na(+) concentrations within narrow limits in collecting duct principal cells  |
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CASP3, CASP9, and MAPK14 interacted with SLIT1-ROBO2 signaling  |
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GSN induces cardiomyocyte hypertrophy and NPPB expression via MAPK14 signaling and GATA4 transcriptional factor activation  |
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IGF1 inhibits cells apoptosis in pulmonary artery smooth muscle cells (PASMC) by activating the MAPK14-NOS2 transduction pathway  |
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negative regulation of MAPK14 mediates the protective effects of SCD on ER homeostasis under distinct stress conditions |
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FNDC5 activated MAPK14 in an AMPK-dependent manner  |
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TLR8, is involved in priming of human neutrophil ROS production by inducing the phosphorylation of NCF1 and MAPK14 and PIN1 is also involved in this process  |
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TNFAIP8L2 inhibited the phosphorylation of AKT1, while promoting the phosphorylation of MAPK14, but had no effect on NFKBIA and ERK pathway  |
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PGF may activate MMP9 via MAPK14 signaling pathway  |
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TNFSF18 could promote Th17 cell differentiation by MAPK14 and STAT3 signaling in autoimmune arthritis  |
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TNFAIP8L2 suppressed breast cancer tumorigenesis, growth and metastasis possibly via regulation of the AKT1 and MAPK14 signaling pathways  |
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CHRM3-induced activation of MAPK14 might contribute to the maintenance of epithelial barrier function through down-regulation of TNF signalling and activation of EGFR  |
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DSG2, beside its adhesion function, regulates intestinal barrier function via MAPK14 signalling  |
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PPP2CA regulated the stem cell factor (SCF)-induced activation of MAPK14/cofilin signaling pathway and subsequent migration of cardiac stem cells (CSCs) by interaction with MAPK14  |
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EXOC3 is involved in the enhancement of cell migration and suppression of apoptosis through the activation of HSPB1, HSPB2 or MAPK14 phosphorylation  |
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MAPK14 mediates epithelial-mesenchymal transition by regulating SUP20TH and SNAI1 in Head and Neck squamous cell carcinoma  |