protein
| p75 nerve growth factor receptors ( |
|
AP kinase-interacting kinase 1 Mnk1 and Mnk2 ( |
|
MAP kinase phosphatase 3, MKP3 ( |
|
PTP-SL and STEP ( |
|
MEK kinase 1, MEKK1 ( |
|
Histone deacetylase 4, HDAC4 ( |
|
MAPK/ERK kinase 1, MEK1 and MAPK/ERK kinase 2, MEK2 |
|
Mitogen-activated protein (MAP) kinase phosphatase 1? MKP-1/CL100 |
|
insulin receptor, IR ( |
|
phosphoprotein enriched in astrocytes 15, PEA15 |
|
hyaluronan binding protein 1, HABP1 ( |
|
caveolin-1, CAV1 ( |
|
MAP kinase Mxi2 ( |
|
RSK1 ( |
|
death-associated protein kinase, DAPK ( |
|
cell division cycle 25 homolog C (S. pombe), CDC25C ( |
|
death effector domain, DED ( |
|
class II, major histocompatibility complex transactivator, CIITA ( |
|
MAPK1/MAPK3 and CEBPB constitute an LH-regulated signaling pathway that controls ovulation- and luteinization-related events |
|
FGF21 effects on chondrocyte function are specific and depend on the normal activity of the FGFR1 and MAPK3 |
| p44 MAPK-deficient mice are viable, fertile, and of normal size but p44 MAPK-/- mice thymocyte maturation beyond the CD4+CD8+ stage was reduced by half and proliferation in response to activation with a monoclonal antibody to the T cell receptor in the presence of phorbol myristate acetate was severely reduced ( | |
mice lacking ERK1 display a dramatic enhancement of striatum-dependent long-term memory ( |
|
disrupted Mapk1/mapk3 in mouse granulosa cells provide in vivo evidence that these kinases are necessary for LH-induced oocyte resumption of meiosis, ovulation, and luteinization |
|
double knock-out newborn pups for Erk1 and Erk2 survived for not more one day, appeared normal just after parturition, displayed intracerebral hemorrhages with varying location and severity, ventricular zones and corpus callosum did not develop properly and nuclear morphology in some brain regions were markedly aberrant ( |
|
double knock-out mice deficient for Erk2 in the central nervous system, with ubiquitous homozygous deletion of Erk1 were neonatally lethal ( |